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Psoriasis fundamentally arises from the interaction between the innate and adaptive immune systems, particularly including T-cells, dendritic cells, and pro-inflammatory cytokines (Schon & Erpenbeck 2018) (Fig

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Fluoxetine-induced androgenic failure impairs the seminiferous tubules integrity and increases ubiquitin carboxyl-terminal hydrolase L1 (Uchl1): possible androgenic control of Uchl1 in germ cell death