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Expert Rev Clin Immunol 2016

The authors conclude that the indicated SOD2 and SOD3 polymorphisms may be genetic risk factors for the susceptibility and progression of vitiligo suggesting the increased activity of SOD isoforms caused by the variant gene polymorphisms may lead to the accumulation of H 2 O 2 in cytoplasmic, mitochondrial and extracellular compartments resulting in melanocyte oxidative damage

According to one study, glutathione helps lessen oxidative stress by either stimulating or reducing the bodys immunological response

These results suggest that the severe fluctuations in calcium signaling increase the susceptibility of substantia nigra neurons to damage and cell death