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Immuno-pathogenesis/hyper-inflammation SARS-CoV-2 infection could disrupt host immune homeostasis, inflict tissue injury, and may persist during the post-recovery phase of COVID-19 survivors and manifest as PASC 210

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In addition, these GPxs have overlapping substrate specificities

However, high glucose-induced NF-B activation, oxidative stress, and inflammation can be ameliorated by a HMGB1 inhibitor, glycyrrhizin, in human renal proximal tubular cells (196)