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The surplus of ROS represents a trigger for programmed cell death processes and promotes the release of pro-inflammatory cytokines, which are also responsible for further increasing ROS levels, thus perpetuating the pathophysiological process of AD

Interestingly, reduced levels of NE and mast cell chymase were observed in neoplastic skin of DPP1 knockout mice, suggesting a role of immune cell serine protease in squamous cell carcinoma development (184)

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*non-contractual picture Use daily after cleansing skin

Effects of DSIP on Biomarkers of Aging, Life Span, and Tumor Incidence in Female Mice